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Shuttle Pharma's Selective HDAC Inhibitor Exhibits ATM Activation and Modulation of ER Expression Resulting in Substantial Growth Inhibition of Estrogen Receptor Positive Breast Cancer Cells, as Reported in PLOS ONE
SHPHShuttle Pharmaceuticals (SHPH) GlobeNewswire News Room·2024-07-19 13:00

Core Insights - Shuttle Pharmaceuticals Holdings, Inc. has published a manuscript demonstrating the effectiveness of its HDAC inhibitor SP-1-303 in inhibiting the growth of estrogen receptor positive breast cancer cells [1][6] - The CEO of Shuttle Pharmaceuticals emphasized the potential of SP-1-303 as a promising therapeutic approach for treating estrogen receptor positive breast cancers through combined targeting of Class I HDACs and ATM [2] Company Overview - Founded in 2012 by faculty members of Georgetown University Medical Center, Shuttle Pharmaceuticals focuses on improving outcomes for cancer patients undergoing radiation therapy [5] - The company's mission is to enhance the effectiveness of radiation therapy while minimizing side effects, aiming to increase cancer cure rates and improve patient quality of life [5] Research and Development - The published manuscript titled "Dual-targeting class I HDAC inhibitor and ATM activator, SP-1-303, preferentially inhibits estrogen receptor positive breast cancer cell growth" was authored by Dr. Mira Jung and Dr. Scott Grindrod [6] - SP-1-303 is a selective Class I HDAC inhibitor that shows direct cellular toxicity in estrogen receptor positive breast cancer and enhances PD-L1 expression, suggesting potential for combination with immune checkpoint blockers [7]